全球变暖可能会增加阻塞性睡眠呼吸暂停的负担
Bastien Lechat1, Jack Manners2, Lucía Pinilla2
1Adelaide Institute for Sleep Health and FHMRI Sleep Health, College of Medicine and Public Health, Flinders University, Adelaide, Australia. bastien.lechat@flinders.edu.au.
Nature communications
|June 16, 2025
概括
较高的环境温度显著增加了阻塞性睡眠呼吸暂停 (OSA) 的风险. 由于温度升温,OSA患病率的增加与健康寿命和工作场所生产率的大幅损失有关.
科学领域:
- 环境健康 环境健康
- 睡眠医学 睡眠医学
- 公共卫生 公共卫生
背景情况:
- 众所周知,高环境温度会减少睡眠时间和睡眠质量.
- 温度对阻塞性睡眠呼吸暂停 (OSA) 严重程度的具体影响仍然在很大程度上未得到量化.
研究的目的:
- 量化24小时环境温度对夜间OSA严重性的影响.
- 在不同的全球变暖情景下估计全球OSA负担和相关的经济影响.
主要方法:
- 分析了超过3.5年的116,620名食品和药物管理局批准的可触摸设备用户的数据.
- 夜间OSA严重程度与24小时环境温度的相关性.
- 预计全球变暖下,与OSA负担相关的福利和生产力损失的估计.
主要成果:
- 在温度较高的夜晚 (第99个比第25个百分位) 观察到患有OSA的概率提高了45%.
- 与全球变暖相关的2023年OSA患病率增加与29个国家的788,198个健康寿命年和300亿美元的工作生产力损失的估计损失有关.
- 预计温度1.8°C高于工业化前的水平,到2100年,OSA负担可能会增加1.2到3倍.
结论:
- 环境温度是影响OSA严重性的重要环境因素.
- 全球变暖通过增加OSA患病率对公众健康构成重大威胁,导致严重的健康和经济后果.
- 应对气候变化的紧急缓解策略是必要的,以减少阻塞性睡眠呼吸暂停的不断升级的负担.
更多相关视频
相关概念视频
Sleep Apnea
227
Sleep apnea is a condition where breathing stops intermittently during sleep, often leading to significant health issues. Each episode can last from 10 to 20 seconds or more and is frequently accompanied by a brief arousal from sleep. This disturbance, largely unnoticed by the individual, can lead to severe daytime fatigue. Commonly, individuals seek help after being informed by their partners about loud snoring and noticeable breathing pauses during sleep.
The condition is more prevalent among...
The condition is more prevalent among...
227
Global Climate Change
24.8K
Throughout its ~4.5 billion year history, the Earth has experienced periods of warming and cooling. However, the current drastic increase in global temperatures is well outside of the Earth’s cyclic norms, and evidence for human-caused global climate change is compelling. Paleoclimatology, the study of ancient climate conditions, provides ample evidence for human-caused global climate change by comparing recent conditions with those in the past.
24.8K
Other Pulmonary Disorders
915
Respiratory disorders encompass a range of conditions with varying levels of severity. Asthma, marked by chronic airway inflammation and hypersensitivity, is one such condition. It can lead to airway obstruction due to factors like bronchial spasms, mucosal edema, increased mucus secretion, or epithelial damage. Asthma triggers are diverse, ranging from allergens to emotional upset, and treatment focuses on both immediate relief through bronchodilators and long-term inflammation suppression.
915
Chronic Obstructive Pulmonary Disease
1.4K
COPD is defined as a heterogeneous lung condition marked by persistent respiratory symptoms such as dyspnea, cough, and sputum production, caused by abnormalities in the airways that cause airflow obstruction.
Smoking is a primary risk factor for COPD, with over 80% of patients having a history of it. Patients typically experience progressive dyspnea or labored breathing, frequent coughing, and recurrent pulmonary infections. Many eventually succumb to respiratory failure, characterized by...
Smoking is a primary risk factor for COPD, with over 80% of patients having a history of it. Patients typically experience progressive dyspnea or labored breathing, frequent coughing, and recurrent pulmonary infections. Many eventually succumb to respiratory failure, characterized by...
1.4K
Hyperpnea and Hyperventilation
1.3K
Hyperventilation refers to a higher-than-normal rate and depth of breathing, often associated with anxiety attacks. This excessive breathing surpasses the body's need to expel CO2, leading to a condition known as hypocapnia - an unusually low level of carbon dioxide in the blood. Hypocapnia can constrict cerebral blood vessels, reducing blood flow to the brain, which may result in dizziness or fainting. Early signs include tingling and muscle spasms in the hands and face, caused by falling...
1.3K
Chronic Obstructive Pulmonary Disease-II: Pathophysiology
3.1K
Chronic Obstructive Pulmonary Disease (COPD) pathophysiology is intricate and multifaceted, involving a complex interplay of physiological processes. Understanding these mechanisms is crucial for effectively managing and treating COPD. Here is an in-depth look at the critical elements in the pathophysiology of COPD:
Chronic Inflammation
Chronic Inflammation
3.1K


