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Updated: Sep 18, 2025

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在催促超痛症之前的抵抗训练通过中央和外围生物标志物调节促进了镇痛效应,在一个类似纤维肌痛的实验模型中

Andrês Valente Chiapeta1, Leandro Licursi de Oliveira2, Luciano Bernardes Leite1

  • 1Laboratory of Exercise Biology, Department of Physical Education, Federal University of Viçosa, Viçosa 36570-900, Brazil.

Life (Basel, Switzerland)
|June 26, 2025
PubMed
概括

在诱导大鼠疼痛之前,抵抗训练 (RT) 预防了机械超痛. 这种干预使骨肌炎症标志物正常化,并提高了thalamic serotonin水平,为纤维肌痛提供了潜在的治疗策略.

关键词:
慢性疼痛是一种慢性疼痛.细胞因子 细胞因子身体炼就是体力炼.血清胺 (serotonin) 是一种神经元.

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