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Updated: Sep 17, 2025

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创伤引发的自身炎症和T细胞自身反应性在创伤后缩的相互作用

Egmont Scola1

  • 1Department of Traumatology, Dietrich-Bonhoeffer-Klinikum Neubrandenburg, affiliated Hospital of Medical School of University Greifswald, Mecklenburg-Vorpommern, Germany.

Frontiers in immunology
|July 3, 2025
PubMed
概括

与损伤相关的分子模式 (DAMPs) 通过激活先天免疫来触发创伤后的无菌炎症. 自然杀手T (NK-T) 细胞和树突细胞 (DCs) 加剧了这种反应,导致潜在的组织损伤和慢性炎症.

关键词:
这是一个AV-shunt.在 NKT 细胞中.自动反应自动活动.与损伤相关的分子模式过度 perfusion 是一种过度 perfusion 的情况.缺氧 缺氧是指缺氧的情况.这是先天免疫力.创伤后伤残症 创伤后伤残症

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