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Updated: Sep 17, 2025

A Simple Protocol for Platelet-mediated Clumping of Plasmodium falciparum-infected Erythrocytes in a Resource Poor Setting
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Published on: May 16, 2013

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抗凝药疗法和改变的组织因子表达有助于预防实验性胎盘和脑疟疾

Alicer K Andrew1,2, Tara C Bracken1, Nicole Nazario-Maldonado2

  • 1Department of Infectious Diseases, College of Veterinary Medicine, and Center for Tropical and Emerging Global Diseases, University of Georgia, Athens, GeorgiaUnited States of America.

PLoS pathogens
|July 3, 2025
PubMed
概括

抗凝剂治疗和降低组织因子 (TF) 保护了对胎盘疟疾 (PM) 和脑疟疾 (CM) 的小鼠模型的严重结果. 这表明,TF在严重疟疾病原发生过程中起着关键作用.

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Warfarin, a prominent vitamin K antagonist family member, exerts its effect by inhibiting the enzyme VKORC1 (vitamin K epoxide reductase complex 1). By hindering this enzyme, warfarin...
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After a fibrin clot is formed, the next step is clot retraction, a vital process facilitated by platelet contractile proteins, such as actin and myosin. These proteins pull the fibrin strands closer together and condense the clot. This action reduces the size of the clot, creating a smaller, denser structure that effectively seals off the damaged vessel. Clot retraction consolidates the clot and helps with wound healing by bringing the edges of the damaged blood vessel closer together.
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