代谢对腿筋肌肉损伤的贡献 在男性的最大,高速偏心运动中
Carsten Schwiete1, Joachim Mester2, Patrick Wahl2,3
1Department of Sports Sciences, Goethe University Frankfurt, Frankfurt am Main, Germany.
Physiological reports
|July 4, 2025
概括
代谢压力导致运动诱导的肌肉损伤 (EIMD) 在高速异常训练期间. 提高有氧能力可以减轻肌肉损伤并促进恢复.
科学领域:
- 运动生理学 运动生理学
- 肌肉生理学 肌肉生理学
- 运动科学 运动科学 运动科学
背景情况:
- 高速度异常训练会导致运动诱导肌肉损伤 (EIMD),主要与机械应变有关.
- 代谢压力的作用在EIMD中,特别是在训练有素的个体中,尚不清楚.
研究的目的:
- 为了研究代谢变化对EIMD在最大偏心腿筋卷曲期间的贡献.
- 在训练有素的运动员中探索代谢标记和肌肉损伤指标之间的关系.
主要方法:
- 30名训练有素的雄性在高速上进行了最大的奇特脚曲.
- 测量了肌肉氧化 (SmO2),气体交换 (VO2,VCO2) 和各种肌肉损伤标志物.
- 线性混合模型分析了代谢参数和损伤标记之间的关联.
主要成果:
- 运动后峰值扭矩显著下降,肌酸激酶 (CK) 在96小时达到峰值.
- 在休息间隔期间增加氧气吸收 (VO2) 预测了较低的CK升高.
- 套件内的更高的有氧需求与更大的力缺陷相关,而SmO2变化并没有显著影响EIMD.
结论:
- 代谢因素在高速异常运动协议中显著导致EIMD.
- 有氧需求和恢复氧气吸收影响肌肉损伤和恢复.
- 提高有氧能力可能对减轻肌肉损伤和改善在苛刻训练中恢复至关重要.
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