微质与树突性脊柱相互作用,并在心脏骤停后的复苏后脑损伤后调节脊柱数量

Jacob M Basak1, Macy Falk1, Andra L Dingman2

  • 1Department of Anesthesiology, University of Colorado Anschutz School of Medicine, Aurora, CO 80045, USA; Neuronal Injury and Plasticity Program, University of Colorado Anschutz School of Medicine, Aurora, CO 80045, USA.

PubMed
概括

在心脏骤停和复苏后,微质细胞增加并导致海马中的突触损失. 在受伤后耗尽微质细胞恢复了突触密度,这表明对认知功能障碍的治疗点.