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Updated: Sep 15, 2025
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Isolation of Primary Murine Brain Microvascular Endothelial Cells
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[S1PR5激活或过度表达通过调节氧化应激来增强小鼠大脑微血管内皮细胞对OGD/R损伤的屏障功能]
Jingxian Wang1, Zijing Ren1, Peiyang Zhou1
1Department of Neurology, Xiangyang First People's Hospital, Hubei University of Medicine, Xiangyang 441000, China.
概括
斯芬哥辛-1-酸盐受体5 (S1PR5) 的激活增强了大脑内皮细胞的活力,并减少了在氧-葡萄糖剥夺/低氧化 (OGD/R) 期间的屏障透性. 这种保护作用涉及减少活性氧物种 (ROS) 和增加抗氧化蛋白质.
科学领域:
- 神经科学是一个神经科学.
- 细胞生物学 细胞生物学
- 生物化学 生化学
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