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Updated: Sep 14, 2025

Author Spotlight: Innovative Techniques for ROS Detection and Implications for Platelet Research
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抑制GPD2通过ROS/NF-κB/P2Y12通路损害了凝血功能

Jiajie Chen1,2,3, Guifeng Xu4, Zhipeng Xie5

  • 1Department of Pharmacy, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Southern Medical University, Guangzhou, Guangdong, China.

Cellular & molecular biology letters
|July 18, 2025
PubMed
概括

GPD2基因 (cg03230175) 的DNA甲基化与冠状动脉心脏病患者的血小板功能受损和出血风险增加有关. 这一发现为个性化抗血小板治疗策略提供了洞察力.

关键词:
通过DNA甲基化.表观遗传机制表观遗传机制在 GPD2 中,GPD2 是指GPD2.在 NF-κBB 中.在P2Y12中,P2Y12在P2Y12中.这就是ROSOS ROS.

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