由肝细胞衍生的Pumilio1丰富的外体通过抑制米-4翻译来抑制HSC激活,从而抑制HSC激活

Zhiping Wan1,2, Xiang Cai1,2, Xiaoquan Liu1,2

  • 1Department of Infectious Diseases, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.

PubMed
概括

与代谢功能障碍相关的脂肪性肝病 (MASLD) 涉及异常的肝细胞交叉交互. 上调Pumilio-1 (PUM1) 通过减少脂质沉积和通过TPM4调节抑制HSC激活来缓解肝损伤.