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Updated: Sep 12, 2025

Detection of Mitochondria Membrane Potential to Study CLIC4 Knockdown-induced HN4 Cell Apoptosis In Vitro
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集群素可以调解免疫性血栓塞缩小症中由亡诱导的分子机制

Tea Stein1, Christina Bitsina1, Michelle Seiler2

  • 1Division of Hematology, Children's Research Center, University Children's Hospital Zurich, Zurich, Switzerland.

Blood vessels, thrombosis & hemostasis
|August 6, 2025
PubMed
概括

通过增加CLU和BAX表达而表明的克拉斯胺中介性亡的损伤可能会导致免疫血小板缩 (ITP) 中的血小板破坏. 这一发现揭示了ITP的病理生理学.

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