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Updated: Jul 18, 2026

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利用患者衍生抗体诱导的微质补充1q表达:针对抗NMDAR脑炎的新疗法

Bingtian Xu1, Yunmeng Bai2, Zhuhe Liu1

  • 1Department of Neurology, Guangzhou First People's Hospital, School of Medicine, South China University of Technology, Guangzhou 510180, China.

Molecular therapy : the journal of the American Society of Gene Therapy
|August 8, 2025
PubMed
概括

抗N-甲基-d-酸盐受体脑炎 (NMDARE) 涉及微质激活. 向补充物C1q,一个关键的调解者,显示了减少NMDARE诱导的大脑损伤的治疗潜力.

关键词:
抗N-甲基-d-酸盐受体脑炎补充 1q 补充 1q 补充补充激活是补充的激活.微质细胞中的微质细胞神经炎症是一种神经炎症.造成神经元损伤.单核RNA测序的一个核.治疗疗法治疗疗法治疗疗法

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Encephalitis ll: Pathophysiology01:26

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Encephalitis is inflammation of the brain parenchyma caused by direct viral invasion or immune-mediated mechanisms triggered by infections or tumors. Both processes lead to neuronal injury, disrupted neurotransmission, and diverse neurological symptoms, often with overlapping clinical and pathological features.Autoimmune EncephalitisIn autoimmune encephalitis, antibodies target neuronal antigens on cell surfaces, synapses, or within neurons. A key example is anti-NMDAR encephalitis, which can...