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Updated: Sep 12, 2025

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巨性Ym1调节γδT细胞衍生IL-17的产生和角质细胞的功能,以调解牛皮样皮肤炎症

Wentao Zhang1,2,3, Fei Li4, Yu Wang2

  • 1National-local Joint Engineering Research Center of Biodiagnostics and Biotherapy, The Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.

Communications biology
|August 9, 2025
PubMed
概括

Ym1基因缺陷通过降低IL-17的产生和角质细胞功能障碍来减少皮肤炎症. Ym1加剧了类似牛皮的皮肤炎,突出了它在这种慢性皮肤疾病中的作用.

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When T cells with CD4 markers are activated, they give rise to two types of effector cells: helper T cells and regulatory T cells. Meanwhile, T cells with CD8 markers differentiate into effector cytotoxic T cells. The differentiation of CD4 T cells into helper T cell subsets, such as Th1, Th2, and Th17 cells, is dependent on the antigen type, antigen-presenting cell, and regulatory cytokines.
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