干旱加剧了levofloxacin的植物毒性:来自Phragmites australis的见解
Yuhang Zong1, Sheng Sheng2, Yin Liu1
1Key Laboratory of Integrated Regulation and Resource Development on Shallow Lakes, Ministry of Education, Hohai University, Nanjing 210098, PR China; College of Environment, Hohai University, Nanjing 210098, PR China.
Journal of hazardous materials
|August 17, 2025
概括
干旱和levofloxacin (LEV) 抗生素污染协同影响Phragmites australis. 综合压力严重抑制生长,破坏植物激素信号传递,降低适应能力.
科学领域:
- 环境科学 环境科学
- 植物生理学 植物生理学
- 分子生物学分子生物学
背景情况:
- 干旱和抗生素污染正在增加环境压力因素.
- 它们对湿地植物的综合影响尚不清楚.
- 利沃素 (LEV) 是一种常见的基诺抗生素.
研究的目的:
- 为了研究干旱和LEV对Phragmites australis的协同植物毒性.
- 了解P. australis对联合压力的生理和分子反应.
- 为了阐明对植物生长,氧化应激和基因表达的影响.
主要方法:
- 一个2x2因数实验被用来模拟干旱和LEV暴露.
- 测量了植物生长,LEV吸收和转移.
- 氧化应激使用综合生物标志物响应指数进行评估.
- 进行了转录组分析,以分析基因表达变化.
主要成果:
- 干旱增加了根部的LEV吸收,但减少了其转移到地表部分.
- 干旱和LEV压力的结合明显抑制了植物生长,并诱导了严重的氧化应激.
- 抗氧化剂系统的综合生物标志物响应指数在所有植物部位显著增加.
- 转录组分析显示了显著的基因表达变化,丰富了植物激素信号转导和MAPK信号通路.
结论:
- 结合干旱和LEV压力对P. australis.表现出协同的植物毒性.
- 这项研究揭示了MAPK-荷尔蒙交叉的干扰,损害了植物的适应能力.
- 结果提供了关于在多种环境压力因素下植物限制的见解.
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