使,IGFBP-3/TMEM219

Maral Amini1, Armin Mokhtariye1, Adel Mohammadalipour1

  • 1Department of Clinical Biochemistry and Isfahan Pharmaceutical Sciences Research Center, Isfahan University of Medical Sciences, Isfahan, I.R. of Iran.

PubMed
概括

通过促进肝细胞亡,IGFBP-3/ TMEM219途径驱动肝纤维化. 针对这种途径为肝纤维化提供了潜在的治疗策略.