在慢性脚不稳定和没有脚不稳定的个体中,下肢肌肉协同作用
Shaghayegh Zivari1, Mohammad Yousefi1, Abbas Farjad Pezeshk1
1Department of Sport Biomechanics, Faculty of Sport Sciences, University of Birjand, Birjand, Iran.
Journal of foot and ankle research
|August 26, 2025
概括
患有慢性脚不稳定 (CAI) 的人在步行开始时表现出改变的肌肉激活模式. 补偿性肌肉策略涉及前骨 (TA),右骨 (RF) 和中骨 (GM_L) 表明全球神经肌肉对脚不稳定的反应.
科学领域:
- 生物力学
- 神经科学
- 运动医学
背景情况:
- 长期脚不稳定 (CAI) 在脚扭伤后损害了姿势控制.
- 步行启动 (GI) 是对中枢神经系统 (CNS) 控制和稳定的敏感指标.
- 肌肉协同提供了关于运动控制神经肌肉策略的见解.
研究的目的:
- 调查 CAI 患者和非 CAI 患者之间的下肢肌肉协同作用的差异.
- 阐明CAI患者在步行开始的关键阶段所采用的神经肌肉策略.
主要方法:
- 一项涉及20名参与者的横截面实验室研究 (10名健康者,10名患有CAI).
- 使用SENIAM指南收集的电肌图 (EMG) 数据,以及基于标记器的运动捕捉系统的动态数据.
- 在OpenSim中使用肌肉骨建模,并在MATLAB中使用HALS进行肌肉协同分析.
主要成果:
- 在健康和CAI组之间肌肉协同的数量没有显著差异.
- 在特定协同作用中观察到肌肉重量的显著差异.
- 在CAI组中,前骨 (TA) 的重量在协同作用1中增加,前骨 (RF) 和中骨 (GM_L) 的重量在协同作用2中增加.
结论:
- 在步行开始时,CAI组通过增加TA,RF和GM_L激活来弥补潜在的长 (PL) 弱点.
- 这些补偿策略表明CAI中神经肌肉控制的改变和运动复杂性的增加.
- 结果表明CAI影响全球运动控制,而不是局部问题,涉及中枢神经系统的适应性策略.
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