预防性,自我应用的光生物调节在治疗口腔粘膜炎:一个高风险的案例研究
Joseph Ryan1, Lee Na Chong2,3,4, Robert Jared Dunn4
1School of Rural Medicine, Charles Sturt University, Orange, NSW, Australia.
Case reports in oncology
|August 29, 2025
概括
在高危癌症患者中,使用904nm和600nm激光的预防性光生物调制 (PBM) 有效地降低了口腔粘膜炎的严重程度. 这种方法有望在抗瘤治疗期间改善生活质量.
科学领域:
- 癌症学
- 生物医学工程
- 激光治疗
背景情况:
- 口腔粘膜炎 (OM) 是癌症治疗的削弱副作用,
- 使用红色和近红外光的光生物调制 (PBM) 是OM的一个有前途的干预措施.
- 最佳的PBM时间和交付方法需要进一步调查.
相关概念视频
01:24Drugs for Peptic Ulcer Disease: Sucralfate as Mucosal Protective Agents
684
In the intricate landscape of the gastric lumen, excessive acid secretion disrupts the natural defense mechanisms, weakening the mucus-bicarbonate barrier. This vulnerability allows pepsin to infiltrate epithelial cells, digesting mucosal proteins and triggering erosion, leading to ulcer formation.
In this scenario, mucosal protective agents like sucralfate play an essential role. Sucralfate, a complex of sulfated sucrose and aluminum hydroxide, demonstrates its usefulness in acidic conditions,...
In this scenario, mucosal protective agents like sucralfate play an essential role. Sucralfate, a complex of sulfated sucrose and aluminum hydroxide, demonstrates its usefulness in acidic conditions,...
684
01:20Drugs for Peptic Ulcer Disease: Prostaglandin Analogs as Mucosal Protective Agents
595
The gastric mucosa produces prostaglandins E2 (PGE2) and prostacyclin (PGI2), crucial in maintaining gastric health. They exert cytoprotective effects, including increasing bicarbonate secretion, releasing protective mucin, reducing gastric acid output, and preventing harmful vasoconstriction. These effects are mediated through various receptors, such as EP1, EP2, EP3, and EP4.
Non-steroidal anti-inflammatory drugs (NSAIDs) can induce peptic ulcers by inhibiting cyclooxygenase, decreasing...
Non-steroidal anti-inflammatory drugs (NSAIDs) can induce peptic ulcers by inhibiting cyclooxygenase, decreasing...
595