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通过调节DRG神经元中的GPR153 / Kcnk16功能,S100A9在神经病痛中发挥作用

Molei Liu1, Dawei Han1, Mingwei Sheng2

  • 1Department of School of Medicine, Nankai University, Tianjin 300071, China.; Department of College of Life Sciences, Nankai University, Tianjin 300071, China.

Cellular signalling
|August 31, 2025
PubMed
概括

一个来自骨髓细胞的分子S100A9, 通过影响背部根中的神经细胞驱动神经病痛. 减少S100A9可以减轻疼痛和炎症,突出其作为治疗点的潜力.

关键词:
背部根结没有.神经性疼痛在 S100A9 中,GPR153

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