受损的氧气消耗动力学和自主功能障碍导致肥胖的年轻男性在持续炼期间疲劳增加
Jatuporn Phoemsapthawee1, Surasak Phoemsapthawee2
1Department of Sports Science, Faculty of Sports and Health Science, Kasetsart University, Nakhon Pathom, Thailand.
Journal of exercise rehabilitation
|September 8, 2025
概括
肥胖会损害运动耐受性,因为它会破坏长时间运动期间氧气吸收 (V·O2) 动力学和自主调节之间的联系. 这表明肥胖个体有氧效率降低.
科学领域:
- 运动生理学 运动生理学
- 自主神经系统功能 自主神经系统功能
- 肥胖问题研究研究
背景情况:
- 肥胖与运动耐受性降低有关,但根本的生理机制尚未完全理解.
- 自主神经系统 (ANS) 调节在运动期间的心血管和代谢调整中起着至关重要的作用.
研究的目的:
- 调查氧气吸收 (V·O2) 动力学是否反映了正常体重与肥胖男性长期中等强度运动期间的自主调节.
- 为了检查不同体重组中V̇O2动力学,心率变化 (HRV) 和感知疲劳之间的关系.
主要方法:
- 一项涉及9名正常体重和9名肥胖男性 (年龄在20-22岁) 的横截面研究.
- 参与者在通风值的70%时,进行了30分钟的恒定负载循环.
- 评估V̇O2动力学 (时间常数 τ,V̇O2漂移斜率) 和休息和运动期间的HRV.
主要成果:
- 与正常体重男性相比,肥胖男性的时间常数 (τ) 显著更长,这表明生理调整速度较慢.
- 肥胖个体在运动期间显示出明显较低的HRV指数,这表明自主反应能力减弱.
- 在正常体重的个体中,V·O2漂移斜率与HRV参数有很强的相关性,肥胖组中没有这种关系.
结论:
- 在长时间炼期间,VO2动力学和自主调节之间的联系在正常体重的个体中被保留,但在肥胖中被破坏.
- 这种自主调节的干扰可能导致有氧效率下降和在肥胖中观察到的运动不耐受性.
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