细胞原体编码的毒性因子Gp05有助于内皮细胞功能障碍和免疫逃避,促进持续的甲基西林耐药黄金葡萄球菌内血管感染

Yi Li1, Adhar C Manna2, Sarah Ibrahim1

  • 1The Lundquist Institute for Biomedical Innovation at Harbor-UCLA Medical Center, Torrance, California, USA.

mBio
|September 8, 2025
PubMed
概括

耐美西林黄金葡萄球菌 (MRSA) 蛋白质Gp05通过破坏内皮细胞和抑制免疫反应,促进内血管感染的持续性. 向Gp05可能为持续的MRSA感染提供新的治疗方法.

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