重复手握强度测试的变化表明,在患有肌痛性脑筋炎/慢性疲劳综合征的患者中,产生的力量低于最大
1Department of Neurology and Center for Translational and Behavioral Neurosciences, University Medicine Essen, University of Duisburg-Essen, Essen, Germany.
European journal of neurology
|September 9, 2025
概括
肌痛性脑筋炎/慢性疲劳综合征 (ME/CFS) 患者的手握强度变化比对照组更高,这表明运动控制问题,而不仅仅是外周功能障碍. 这一挑战使得握力成为ME/CFS的唯一生物标志物.
科学领域:
- 神经学 神经学
- 生物标志物 生物标志物
- 慢性疲劳研究 慢性疲劳研究
背景情况:
- 手握强度被提议作为肌痛性脑膜炎/慢性疲劳综合征 (ME/CFS) 的生物标志物.
- 这假定在ME/CFS中存在外围神经肌肉功能障碍.
- 另一种假设认为,更高阶的运动控制变化有助于衰弱.
研究的目的:
- 分析ME/CFS患者和健康对照者的手握强度变化.
- 要调查是否改变了运动控制,而不是外周功能障碍,解释了ME/CFS中的力量缺陷.
主要方法:
- 从105名ME/CFS患者和66名对照患者的连续握手强度测量结果的特设分析.
- 在手握力度变化系数 (CV) 的比较.
- 对具有正常疲劳指数的参与者的子组分析.
主要成果:
- 与健康对照组相比,ME/CFS患者的CV显著增加.
- 在女性ME/CFS患者中,即使具有正常的疲劳能力,心血管的升高仍然存在.
- 在正常疲劳的男性ME/CFS患者中,CV没有发现显著差异.
结论:
- 在ME/CFS中增加的手握强度变化表明涉及更高阶运动控制缺陷.
- 这一发现挑战了将握力作为ME/CFS中外周功能障碍的生物标志物的使用.
- 功能性弱点可能源于慢性失调的背景下自我效能降低.
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