酸改善TLR4介导的巨细胞激活,通过不可逆转地与百氧化素1结合,以抑制其二分化和分泌

Jie Gao1, Jin Yan1, Xingwang Zu1

  • 1State Key Laboratory of Medicinal Chemical Biology, College of Pharmacy and Tianjin Key Laboratory of Molecular Drug Research, Nankai University, Tianjin 300353, China.

概括

费鲁酸 (FA) 协同结合到百氧化素1 (PRDX1),减少其炎症信号传递. 这种FA的新机制针对与损伤相关的分子模式 (DAMP) 来缓解炎症.