IGF2BP3介导的NTSR1 m6A甲基化增强了对虹膜素的耐药性和LUAD恶性进展

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Hao Ding1, Xuan Zhu1, Yongquan Pan1

  • 1Affiliated People's Hospital of Jiangsu University, Zhenjiang, China.

American journal of respiratory cell and molecular biology
|September 23, 2025
PubMed
概括

神经激素受体1 (NTSR1) 促进肺腺癌 (LUAD) 的进展和对义诺他干的耐药性. 它的稳定性由IGF2BP3介导的m6A甲基化增强,这表明LUAD治疗的治疗标.

关键词:
在 IGF2BP3BP3 中.氨酸是一种氨酸.这是一个NTSR1规则.肺部腺癌的癌症.m6A甲基化 的情况.

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