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通过FIT2介导的脂质滴生物合成和FABP5的协调抑制M1巨细胞极化

Yi-Han Liu1, Bei Li1, Yuan-Xing Zhang2,3

  • 1State Key Laboratory of Bioreactor Engineering, East China University of Science and Technology, Shanghai 200237, China.

Zoological research
|September 29, 2025
PubMed
概括

脂质滴滴调节巨细胞的免疫反应. 受FIT2和FABP5影响的脂肪滴量的增加抑制了M1极化,损害了抗菌防御.

关键词:
在FABP5中,FABP5是FABP5中的一个.在FIT2中,FIT2是FIT2.脂质滴滴是什么 脂质滴滴是什么巨细胞的两极分化

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Lipid Catabolism01:25

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Triglycerides serve as crucial long-term energy storage molecules in microorganisms, providing a dense source of metabolic energy. Their breakdown is mediated by lipases, which hydrolyze triglycerides into glycerol and free fatty acids. Each of these components follows distinct metabolic pathways, ultimately contributing to ATP synthesis and cellular energy homeostasis.Glycerol MetabolismGlycerol, released from triglyceride hydrolysis, is phosphorylated by glycerol kinase to form...
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