纠正:停止压缩内皮层气溶化疗 (PIPAC) 的原因:为了改善未来的患者选择,进行了一项回顾性研究
PloS one
|October 17, 2025
概括
这项研究纠正了先前发表的一篇文章. 校正确保了科学记录的准确性,并为未来的研究提供了适当的引用.
科学领域:
- 科学出版科学出版
- 学术传播学术交流
背景情况:
- 确保发表的科学文献的完整性至关重要.
- 准确的数字物体标识符 (DOI) 对于引用研究至关重要.
研究的目的:
- 纠正已发表文章的数字物体标识符 (DOI) 中的错误.
- 维护科学研究的准确性和可检索性.
主要方法:
- 对该文章发布了纠正通知.
- 错误的DOI被确定并纠正.
主要成果:
- 文章的DOI已更新到正确的值.
- 校正确保了正确的归因和对研究的访问.
结论:
- 这次纠正符合科学出版的标准.
- 准确的DOI促进了可靠的科学话语和知识传播.
相关概念视频
Pneumothorax-II
897
Pneumothorax is a medical condition defined by the buildup of air in the pleural space between the lungs and the chest wall. This accumulation of air can lead to partial or complete lung collapse, resulting in a range of clinical manifestations. Understanding the clinical presentation and effective management strategies is crucial for healthcare professionals in providing timely and appropriate care to individuals with pneumothorax.
Clinical Manifestations:
Clinical Manifestations:
897
Treatment for Pulmonary Arterial Hypertension: Prostacyclin Receptor Agonists
457
Prostacyclin receptor agonists are a class of therapeutic agents integral to managing pulmonary arterial hypertension (PAH). These drugs operate by mimicking the action of prostaglandin I2, or PGI2, a naturally occurring compound in the body.
These agonists bind to the IPR receptor situated on the plasma membrane of the pulmonary artery smooth muscle cells. This binding triggers a cascade of reactions known as the GS-AC-cAMP-PKA pathway. This pathway results in the relaxation of smooth muscle...
These agonists bind to the IPR receptor situated on the plasma membrane of the pulmonary artery smooth muscle cells. This binding triggers a cascade of reactions known as the GS-AC-cAMP-PKA pathway. This pathway results in the relaxation of smooth muscle...
457


