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通过NOX2途径对血小板活性进行PTH驱动的调节,用于术后的低甲状腺症

Alessandra D'Amico1, Gaia Tabacco2, Cristina Nocella3

  • 1IRCCS Neuromed, Pozzilli, Italy; Department of Health and Life Sciences, European University of Rome, Via degli Aldobrandeschi 190, 00163, Rome, Italy.

Redox biology
|October 18, 2025
PubMed
概括

术后慢性缺甲状腺症 (HypoPT) 通过增强血小板激活和氧化应激增加心血管风险. 副甲状腺激素 (PTH) 治疗可能会通过NOX2-依赖性途径恶化这些影响,需要密切监测.

关键词:
甲状腺功能下降症 甲状腺功能下降症氧二氧化 (NOX2) 是一种氧化应激是一种氧化应激.PTHTH PTH 这是一个很好的方法.血小板激活是指血小板激活.氧化还原信号的信号.

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