视垂体和视通道病变伪装成玻璃眼:突出诊断挑战
Priti Singh1, Samendra Karkhur1, Vidhya Verma1
1Ophthalmology, All India Institute of Medical Sciences, Bhopal, IND.
Cureus
|October 28, 2025
概括
压缩性视神经病变可以模仿玻璃眼,导致误诊. 早期的神经成像对于检测内病变和预防视力丧失至关重要.
科学领域:
- 眼科医生 眼科 眼科
- 神经学 神经学
- 放射学 放射学是一门学科.
背景情况:
- 玻璃眼光神经病变和压缩性光神经病变具有相似的视野缺陷和光盘变化.
- 区分这些疾病对于适当的患者管理至关重要.
研究的目的:
- 为了识别诊断挑战在区分玻璃眼光神经病与压缩性光神经病.
- 强调神经成像在诊断模仿青光眼的内病变中的作用.
主要方法:
- 六名患者的回顾性病例系列,最初怀疑患有青光眼.
- 对临床发现,视野缺陷,光盘外观和神经成像结果的审查.
主要成果:
- 所有患者都呈现了光盘缩/光和视野缺陷,类似于玻璃眼.
- 非典型的特征,如垂直午线节省的VFD,同名的半视障碍和不成比例的圆盘白,表明非黄昏病的病因.
- 神经成像显示了垂体宏腺瘤 (4名患者),脑膜瘤 (1名患者) 和视通道损伤 (1名患者).
结论:
- 压缩性视神经病变经常模仿玻璃眼,可能导致诊断延迟和不正确的治疗.
- 识别非典型的视野模式和光盘发现是关键.
- 及时的神经成像对于诊断压缩性病变和避免不可逆转的视力丧失至关重要.
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