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Updated: Jan 12, 2026

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抑制NRF2激活甲基醇-氨酸缩剂诱导的氧化应激和巨细胞和脂肪细胞之间的炎症性副膜相互作用

Hee-Weon Lee1,2, Min Ji Gu1, Donghwan Kim1

  • 1Korea Food Research Institute, Jeonbuk 55365, Korea.

Preventive nutrition and food science
|November 3, 2025
PubMed
概括

甲基酸二聚物 (MOLD) 通过增加先进的糖化终产物 (AGEs) 来触发炎症和脂肪生成. 激活NRF2减轻了这些MOLD诱导的影响,这表明它在代谢障碍中的作用.

关键词:
先进的糖化最终产品.这是一种炎症炎症炎症炎症.巨细胞是什么?巨细胞是什么?肥胖 肥胖 肥胖 肥胖 肥胖 肥胖 肥胖 肥胖这些受体是受体受体.

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