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Updated: Jan 12, 2026

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在创伤性脑损伤后,NSUN6通过通过m5C修饰增强HMGB1表达来促进神经元轴突再生

Lei Wang1, Quanming Zhou2, Jianning Chen2

  • 1Department of Neurosurgery, The Second Affiliated Hospital of Fujian Medical University, Quanzhou, Fujian, China; Department of Neurosurgery, The Affiliated Hospital of Putian University, Putian, Fujian, China.

Brain research bulletin
|November 6, 2025
PubMed
概括

NOP2/SunRNA甲基转移酶6 (NSUN6) 在创伤性脑损伤 (TBI) 后促进轴突再生. NSUN6通过m5C甲基化增强高流动性组盒1 (HMGB1) 表达,改善神经功能,并为TBI提供治疗点.

关键词:
这就是HMGB1的特点.在M5C中,它是M5C.在NSUN6中神经元轴突再生的神经元轴突再生创伤性脑损伤是一种创伤性脑损伤.

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In the CNS, neurogenesis, the birth of new neurons from stem cells, is limited to the hippocampus in adults. In other regions of the brain and spinal cord, neurogenesis is almost non-existent due to inhibitory influences from neuroglia, especially oligodendrocytes, and the absence of growth-stimulating cues. The myelin produced by oligodendrocytes in the CNS inhibits neuronal regeneration. Furthermore, astrocytes proliferate rapidly after neuronal damage, forming scar tissue that physically...
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