NF-κB/IL-6轴驱动在缺乏水分的干眼中受损的角膜伤口愈合

Cuipei Lin1,2, Jiaxin Wu1,2, Yuxin Jing1,2

  • 1International Ocular Surface Research Center, Institute of Ophthalmology, and Key Laboratory for Regenerative Medicine, Jinan University, Guangzhou, China.

Frontiers in immunology
|November 10, 2025
PubMed
概括

干眼疾病通过激活NF-κB/IL-6炎症通路,损害角膜愈合. 针对这些途径进行治疗,如CAPE或抗IL-6抗体,可以改善角膜上皮和神经修复.