由于在微血管减压期间的手术内 supratentorial 亚硬质血瘤引起的急性脑部胀:一个病例报告
Jia Shi1, Jiachao Cao1, Qiang Zhou1
1Department of Neurosurgery, The Third Affiliated Hospital of Soochow University, Changzhou, China.
Frontiers in surgery
|November 10, 2025
概括
神经血管压缩的微血管减压 (MVD) 手术可能会导致罕见的手术内静脉血瘤. 迅速治疗这种并发症对于预防严重的大脑胀和确保患者康复至关重要.
科学领域:
- 神经外科 神经外科
- 神经学 神经学
- 手术并发症 手术并发症
背景情况:
- 微血管减压 (MVD) 是神经血管压缩综合征的黄金标准治疗方法.
- 出血是一种关键的手术并发症,手术内 supratentorial 腹腔内血瘤 (SDH) 是不常见的,但复杂的管理.
- 在MVD期间缺乏用于管理手术内SDH的标准化协议.
研究的目的:
- 为了检查MVD期间的急性脑部胀的呈现和管理,二次到手术内SDH.
- 强调对这种罕见但可能致命的并发症保持警的重要性.
- 讨论预防和管理严重脑和脑的挑战.
主要方法:
- 病例报告详细介绍了三神经疼痛中MVD期间的手术内 supratentorial SDH.
- 专注于有效的内压力管理和预防二次脑损伤.
- 对手术内SDH的呈现和管理策略的文献审查.
主要成果:
- 尽管发生了手术内SDH,但MVD的成功完成.
- 有效管理急性脑部胀和内压力.
- 对患者有利的术后康复.
结论:
- 在MVD期间的手术内SDH,虽然很少见,但可能导致严重的大脑胀和脑.
- 警和迅速,有效的管理对于防止灾难性的结果至关重要.
- 对这种并发症的标准化管理协议进行进一步的研究是有必要的.
相关概念视频
Hemorrhagic Stroke l: Introduction
41
A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
41
Hemorrhagic Stroke ll: Pathophysiology
57
A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
57
Brain Abscess l: Introduction
48
A brain abscess is a focal, intracerebral infection characterized by a localized collection of pus within the brain parenchyma, resulting from microbial invasion and the body’s inflammatory response. It progresses through stages: early and late cerebritis, followed by early and late capsule formation, reflecting tissue destruction, immune response, and eventual encapsulation.Etiology and PathogenesisCausative organisms vary with source and host factors, often involving polymicrobial...
48
Increased Intracranial Pressure l: Introduction
31
Intracranial hypertension is a sustained elevation of intracranial pressure (ICP) above 22 mm Hg. In supine adults, normal ICP is ~7–15 mm Hg.The rigid, nonexpandable cranium contains three components—brain tissue, blood, and cerebrospinal fluid (CSF)—that total ~1,700 mL in a typical adult: 1,400 mL brain (~80%), 150 mL blood (~10%), and 150 mL CSF (~10%). According to the Monro–Kellie doctrine, total intracranial volume is effectively fixed. When one component...
31
Cerebral Edema l: Introduction
35
Cerebral edema is a pathological increase in brain water content that disrupts intracranial pressure regulation and impairs neurological function. Because the cranial vault is rigid, even modest increases in tissue volume can compromise cerebral perfusion, distort neural structures, and initiate secondary injury. Cerebral edema develops through four principal mechanisms: vasogenic, cytotoxic, interstitial, and ionic.Vasogenic EdemaVasogenic edema arises from disruption of the blood–brain...
35
Cerebral Edema ll: Pathophysiology
31
Vasogenic edema is a major form of cerebral edema characterized by abnormal accumulation of fluid in the brain’s extracellular space due to disruption of the blood–brain barrier (BBB). The BBB is a specialized structure composed of endothelial cells connected by tight junctions, supported by astrocytic endfeet and a basement membrane. Under normal conditions, it tightly regulates the movement of ions, proteins, and solutes between the bloodstream and brain parenchyma. When this...
31


