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Updated: Jan 6, 2026

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在叶酸缺乏引起的神经管缺陷中,Gcm1调节左侧1表达

Zhihua Cao1,2, Lu Li1, Xiaolong Gu1

  • 1Department of Biochemistry and Molecular Biology, College of Basic Medicine, Shanxi Key Laboratory of Birth Defect and Cell Regeneration, MOE Key Laboratory of Coal Environmental Pathogenicity and Prevention, Shanxi Medical University, Taiyuan, 030001, PR China.

Molecular neurobiology
|November 11, 2025
PubMed
概括

叶酸缺乏会通过基因素乙化增加缺失1 (Gcm1) 表达的质细胞,导致神经管缺陷 (NTDs). 这种Gcm1-Lef1通路为NTD提供了潜在的治疗点.

关键词:
乙化 乙化是一种这是一种CREB结合蛋白.缺少的质细胞 1 缺少的质细胞淋巴细胞增强剂结合因子1神经管缺陷的神经管缺陷

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