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Updated: Jan 11, 2026

Mechanism of Kemeng Fang's Inhibition of Podocyte Apoptosis in Rats with Membranous Nephropathy through the PI3K/AKT Signaling Pathway
07:15

Mechanism of Kemeng Fang's Inhibition of Podocyte Apoptosis in Rats with Membranous Nephropathy through the PI3K/AKT Signaling Pathway

Published on: August 23, 2024

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一个CPEB2/ANGPTL3的反循环促进了在性综合征中细胞损伤的进展

Xin Zheng1,2,3, Shuibao Chen2,3, Yizhong Chen4

  • 1College of Clinical Medicine for Obstetrics & Gynecology and Pediatrics, Fujian Medical University, Department of Nephrology, Rheumatology and Immunology, Fujian Children's Hospital, 966 Heng Yu Road, Fuzhou, 350014, Fujian Province, China.

Scientific reports
|November 11, 2025
PubMed
概括

这项研究揭示了细胞质多基化元素结合蛋白2 (CPEB2) 和血管蛋白样-3 (ANGPTL3) 之间的反循环,该反循环驱动了瘤综合征 (NS) 中的 podocyte 损伤. 针对这个循环可能为NS提供新的治疗策略.

关键词:
ANGPTL3 在线观看在CPEB2中,我们可以使用CPEB2.脏病综合征 - 脏病综合征足细胞损伤的损伤

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