FBXO5驱动肝细胞癌的进展,是茶叶多中介抑制的目标

Xinyu Wang1, Dandan Yang1, Jiao Wang1

  • 1Bengbu Medical University Key Laboratory of Cancer Research and Clinical Laboratory Diagnosis, Bengbu Medical University, Bengbu, China.

PubMed
概括

在肝细胞癌 (HCC) 中,FBXO5作为瘤基因起作用. 茶叶多醇 (TPs) 通过降低FBXO5的调节来抑制HCC的进展,这表明TPs是治疗HCC的潜在治疗剂.

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