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Updated: Jan 11, 2026

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Enhancement of Apoptotic and Autophagic Induction by a Novel Synthetic C-1 Analogue of 7-deoxypancratistatin in Human Breast Adenocarcinoma and Neuroblastoma Cells with Tamoxifen

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一种新合成的化合物通过向线粒体功能障碍和TP53/BCL-2/SEMA3通路来诱导前列腺癌细胞的亡

Neslihan Meriç1, Ezgi Kar2, Fatih Kar3

  • 1Department of Molecular Biology and Genetics, Faculty of Engineering and Natural Sciences, Kütahya Health Sciences University, Kütahya, Turkey.

Biochemical and biophysical research communications
|November 19, 2025
PubMed
概括

一种新型的醇衍生物有效地降低了前列腺癌细胞活力,并诱导了亡. 该化合物选择性地向癌细胞,调节炎症信号,并支持其在瘤学中的治疗潜力.

关键词:
4 - 甲基thiazole 的使用情况.细胞灭亡 (apoptosis) 是一种死亡的过程.细胞循环的细胞周期炎症信号传递是炎症信号.线粒体功能障碍 线粒体功能障碍的PC-3细胞.这就是SEMA3A的意义.

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