骨髓细胞衍生的NLRP3可用于二氧化引起的肺炎和病理

Kristian T Barry1,2, Christopher M Harpur1,2, Rebecca L Ambrose1,2

  • 1Centre for Innate Immunity and Infectious Disease, Hudson Institute of Medical Research, Clayton, VIC, Australia.

Immunology and cell biology
|November 20, 2025
PubMed
概括

骨髓细胞NLRP3炎症酶对病的发展并非必不可少. 在这些细胞中删除NLRP3并没有阻止小鼠的氧化物诱导的肺炎,纤维化或结节形成.

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