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阿斯特拉加勒斯的多糖阻碍了宫癌通过向NR3C2和激活SLC40A1来阻止免疫逃生

Wenzhi Liu1, Lu Zhang2, Yixin Wang3

  • 1Medical Education Department, Changzhou Hospital of Traditional Chinese Medicine, Changzhou, 213003, Jiangsu, PR China.

Biochimica et biophysica acta. Molecular basis of disease
|November 26, 2025
PubMed
概括

阿斯特拉加卢斯多糖 (APS) 通过向NR3C2和激活SLC40A1.1.2来抑制宫癌免疫逃生. 这种机制增强了CD8+T细胞的功能,为宫癌提供了潜在的治疗策略.

关键词:
阿斯特拉加勒斯的多糖化物.宫癌是发生在宫癌的原因之一.免疫逃脱是为了逃脱免疫.在NR3C2中,它是NR3C2.在SLC40A1中,它是SLC40A1的.

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