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通过PPAR-γ/NRF2轴,IL-38通过调节GPX3介导的抗氧化防御来减轻血管化

Lei Zhu1, Qiang Wang2, Bingliang Shang2

  • 1Department of Cardiovascular Surgery, Xijing Hospital, The Fourth Military Medical University, Xi'an, China.

Cellular and molecular life sciences : CMLS
|November 27, 2025
PubMed
概括

介质素-38 (IL-38) 通过减少氧化应激来抑制血管化. 这一发现揭示了IL-38作为心血管疾病的潜在治疗点.

关键词:
在GPX3中使用GPX3.它们的IL-38是IL-38.这就是NRF2的NRF2.氧化应激是一种氧化应激.在PPAR-γ中使用PPAR.血管化是因为血管化.

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