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Updated: Jan 6, 2026

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塞尔宾A3k缺乏改善了实验性2型糖尿病

Isaac González-Soria1,2,3, Esmeralda Palacios-Brito1,4, Dulce Gómez-Trujillo1,2

  • 1Universidad Nacional Autónoma de México, Mexico City, Mexico.

Cellular and molecular life sciences : CMLS
|December 2, 2025
PubMed
概括

塞尔宾A3k缺乏保护小鼠免受肥胖和2型糖尿病 (T2D) 的并发症. 准SerpinA3k可能为T2D等代谢疾病提供新的治疗策略.

关键词:
脂肪细胞是一种脂肪细胞.胰岛素耐药性 胰岛素耐药性是一种肥胖问题 肥胖问题功能障碍 功能障碍 功能障碍

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