相关实验视频

Updated: Jan 9, 2026

Drug-Induced Senescence in Liver Cells Promotes M2 Macrophage Polarization: Implications for Tyrosine Kinase Inhibitor-Associated Hepatotoxicity
09:32

Drug-Induced Senescence in Liver Cells Promotes M2 Macrophage Polarization: Implications for Tyrosine Kinase Inhibitor-Associated Hepatotoxicity

Published on: October 17, 2025

406

乳糖驱动的KRT19通过抑制细胞衰老促进非小细胞肺癌的进展

Cai Zhang1, Yue Du2, Yangyang Ji2

  • 1Department of Clinical Laboratory, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, 450052, China. zhangcai9394@zzu.edu.cn.

Journal of experimental & clinical cancer research : CR
|December 4, 2025
PubMed
概括

这项研究表明,KRT19通过覆盖细胞衰老来促进非小细胞肺癌 (NSCLC). 抑制KRT19可以增强抗瘤免疫力,并且在NSCLC患者的组合疗法中表现有前途.

关键词:
细胞衰老 细胞衰老KRT19 在线观看乳化 乳化 乳化非小细胞肺癌是非小细胞肺癌.在P2121中,P21是指P21.在Ubiquitination中使用.

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