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通过Akt/Nrf2通路调制,在脑缺血-再输液损伤中通过ZNF655介导的神经保护

Wanyi Wei1, Zongkai Wu2, Yuehong Yue2

  • 1Department of Neurology, Hebei Medical University, Shijiazhuang 050017 Hebei Province, China; Department of Neurology, Hebei General Hospital, Shijiazhuang 050051 Hebei Province, China.

Brain research
|December 6, 2025
PubMed
概括

指蛋白655 (ZNF655) 通过激活Akt/Nrf2通路来保护大脑缺血-再输液损伤 (CIRI). 在CIRI模型中,抑制ZNF655可降低亡,氧化应激和炎症.

关键词:
这就是Akt/Nrf2的意思.细胞灭亡 (apoptosis) 是一种死亡的过程.大脑缺血再输血损伤炎症 炎症是一种炎症.氧化应激是一种氧化应激.这就是ZNF6555.

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