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Updated: Jan 8, 2026

Flow Cytometric Analysis for Identification of the Innate and Adaptive Immune Cells of Murine Lung
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天生的2型淋巴细胞触发了膜巨细胞中的炎症开关

Stijn Verwaerde1, Jean-François Hastir2, Sjoerd T T Schetters1

  • 1Laboratory of Immunoregulation and Mucosal Immunology, VIB-UGent Center for Inflammation Research, Ghent 9052, Belgium; Department of Internal Medicine and Pediatrics, Ghent University, Ghent 9000, Belgium.

Immunity
|December 12, 2025
PubMed
概括

组织寄居的膜巨细胞 (trAMs) 通常调节免疫力,但在过敏反应期间可以变得有炎症作用. 从PPARγ切换到IRF4可以重新编程Trams,从而导致肺病理.

关键词:
在 ILC2 中.在 IRF4 基础上,IRF4 是一个 IRF4 项目.在 PPARγγ 中.另类激活的巨细胞.膜膜巨细胞的巨细胞喘 喘 是一种化学物质 (chemokines) 是一种化学物质.巨细胞的利基多核巨细胞多核巨细胞第二种类型的免疫.

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