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微粒对内皮受体CD36的接触触发了CO中毒中神经炎症和淋巴功能障碍

Stephen R Thom1, Awadhesh K Arya1, Abid R Bhat1

  • 1Department of Emergency Medicine, University of Maryland School of Medicine, Baltimore, Maryland, United States.

Journal of neurophysiology
|December 15, 2025
PubMed
概括

一氧化碳 (CO) 中毒会通过通过CD36粘附于大脑血管的循环微粒引起神经炎症,引发大脑损伤的循环. 阻止CD36可以防止这种CO诱导的神经炎症级联.

关键词:
克雷弗洛克斯 (Cre/flox) 是一个流动的生物.它们的内皮质 (endothelium).这些微粒是微粒.神经炎症是一种神经炎症.作为一个中性恋者.

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