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COVID-19感染和事件高血压的风险:一个病例控制时间到事件的研究

Jeremy David Goldhaber-Fiebert^1,2, Shannon C Phillips^1,2, Kimberley D Lucas³

¹Department of Health Policy, Stanford Medical School, Stanford University, Stanford, CA, 94305 USA.

American journal of epidemiology

|December 22, 2025

概括

COVID-19感染显著增加高血压风险,特别是在BMI,血压和年龄较高的个体中. 这项研究证实了SARS-CoV-2和新发性高血压之间的联系.

科学领域:

流行病学流行病学
传染性疾病传染性疾病
心血管健康心血管健康

背景情况:

之前对COVID-19和高血压 (HTN) 的研究缺乏纵向数据和少数群体的代表性.
SARS-CoV-2 感染和随后的HTN 发病率之间的关系需要进一步调查,特别是在脆弱人群中.

研究的目的:

为了检查COVID-19感染和事件高血压在一个大规模的监禁队列之间的关联.
解决先前研究的局限性,包括纵向数据,诊断信息和多种人群.

主要方法:

一项针对加利福尼亚州监狱中39746名被监禁人员的病例控制时间对事件的研究.
纳入标准:没有先前的HTN,没有血压药物,流行前和后的BMI和BP测量,以及COVID-19测试.
使用多变量Cox比例危险模型,调整相关共变量并评估混因素.

主要成果:

COVID-19 感染与高血压发生率显著增加有关 (HRR: 1.44 [1.32-1.57]).
当考虑相互作用时,这种关联更强烈 (HRR:2.05 [1.50-2.79]).
在BMI较高,血压升高和年龄较大的人群中观察到HTN风险增加.

结论:

感染COVID-19是发展高血压的重要危险因素.
这些发现是强有力的,并不能通过增加感染后的诊断测试来解释.
这项研究强调了COVID-19的长期心血管影响,特别是在特定的人口和临床子组.

关键词:

在 COVID-19 疫情中,高血压血压升高血压升高在高血压的高血压.事件发生率事件发生率风险因素风险因素这就是SARS-CoV-2病毒.

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