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Updated: Jan 8, 2026

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升高的miR-17-5p通过向巨细胞中的MAP3K2来促进菌根细菌的免疫逃避

Md Shoykot Jahan1,2, Jinlan Yang1,2, Yiyue Tang3

  • 1School of Life Sciences, Central South University, Changsha, China.

Frontiers in immunology
|December 22, 2025
PubMed
概括

微RNA-17-5p通过抑制巨细胞的免疫反应促进了Mycobacterium avium的生存. 结核病患者的升高miR-17-5p表明其作为诊断生物标志物和治疗点的潜力.

关键词:
艾维乌姆 (M. Avium) 是一个著名的作家.在MAP3K2K2中,您可以使用MAP3K2.结核病菌菌菌的结核病菌的结核病菌.诊断标志物是一个诊断标志物.这是一种炎症炎症炎症炎症.在 miR-17-5p 中.

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