COVID-19作为沃尔克曼缺血收缩发展的促成因素:一个病例报告
Tomas Maciulaitis1, Patricija Glovackaite2, Mindaugas Minderis3
1Faculty of Medicine, Institute of Clinical Medicine, Clinic of Rheumatology, Orthopedics, Traumatology and Reconstructive Surgery, Vilnius University, Vilnius, LTU.
Cureus
|December 25, 2025
概括
沃尔克曼缺血收缩症 (VIC) 是隔间综合征的一种严重并发症. 这一案例突显了COVID-19期间的诊断挑战,强调了早期识别以防止不可逆转的肌肉损伤.
科学领域:
- 整形外科 整形外科 整形外科
- 血管外科 血管外科
- 传染性疾病 传染性疾病
背景情况:
- 沃尔克曼缺血性收缩症 (VIC) 是由于未经治疗的急性隔间综合征引起的,导致不可逆转的曲形.
- 随着COVID-19的流行,诊断和管理像隔间综合症这样的疾病面临着独特的挑战.
- 错误诊断和延迟治疗可能导致严重的后果,包括肌肉死.
研究的目的:
- 在错误诊断后在COVID-19隔离期间报告发生VIC病例.
- 讨论COVID-19在VIC病理生理学中的潜在作用.
- 强调及时识别隔间综合征的重要性,即使没有创伤.
主要方法:
- 一个55岁的男性的病例报告,前臂 flexor 肌肉收缩.
- 对临床表现,诊断过程和治疗干预措施的审查.
- 讨论潜在的COVID-19相关的病理生理机制.
主要成果:
- 在COVID-19隔离期间,患者在最初错误地将症状归因于细胞炎后发展出VIC.
- 延迟诊断和干预导致肌肉死亡和固定曲形.
- 手术减压,脱bridement和神经溶解导致部分功能恢复.
结论:
- 不管是什么创伤,早期识别隔间综合征对于预防VIC至关重要.
- COVID-19可能通过诸如细胞因子介导的胀和内皮功能障碍等机制促进VIC的发展.
- 流行病期间的诊断延迟和治愈障碍需要提高临床警.
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