Jove
Visualize
联系我们
JoVE
x logofacebook logolinkedin logoyoutube logo
关于 JoVE
概览领导团队博客JoVE 帮助中心
作者
出版流程编辑委员会范围与政策同行评审常见问题投稿
图书馆员
用户评价订阅访问资源图书馆顾问委员会常见问题
研究
JoVE JournalMethods CollectionsJoVE Encyclopedia of Experiments存档
教育
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab Manual教师资源中心教师网站
使用条款与条件
隐私政策
政策

相关概念视频

Blood Studies for Cardiovascular System I: Cardiac Biomarkers01:20

Blood Studies for Cardiovascular System I: Cardiac Biomarkers

749
Cardiac biomarkers are enzymes, proteins, and hormones released into the blood when cardiac cells are injured. They are powerful tools for triaging.
The essential diagnostic tools for detecting myocardial necrosis and monitoring individuals suspected of having acute coronary syndrome (ACS) include:
Troponins
Troponins, particularly cardiac troponins I and T, are the most precise and sensitive markers of myocardial injury. They are detectable within 4-6 hours of myocardial injury and remain...
749
Blood Studies for Cardiovascular System II: CRP, Hcy, and Cardiac Natriuretic Peptide Markers01:19

Blood Studies for Cardiovascular System II: CRP, Hcy, and Cardiac Natriuretic Peptide Markers

516
Cardiac biomarkers are critical in diagnosing, prognosing, and managing cardiovascular diseases. Routine measurement of specific biomarkers such as B-type natriuretic peptide (BNP), C-reactive protein (CRP), and homocysteine (Hcy) is common practice in clinical settings to evaluate heart function and predict cardiovascular events.
These markers indicate stress or strain on the heart muscle:
Natriuretic Peptides (BNP)
Cardiac myocytes produce these hormones in response to ventricular stretching...
516

您也可能阅读

相关文章

通过共同作者、期刊和引用图与本文相关的文章。

排序
Same author

Impact of plasma pTau181 levels on clinician diagnostic confidence and management in memory and cognition clinics: A multi-site before-and-after study.

Alzheimer's & dementia (Amsterdam, Netherlands)·2026
Same author

Neuropsychologists' Engagement With and Cognitive Assessment in Parkinson's Disease: A Mixed Methods Australian Investigation.

Journal of geriatric psychiatry and neurology·2026
Same author

White matter hyperintensities are associated with locus coeruleus atrophy and astrocytic β<sub>2</sub>-adrenergic receptor expression.

Alzheimer's & dementia : the journal of the Alzheimer's Association·2026
Same author

Feasibility and Acceptability of a Remote Sleep-Dependent Memory Assessment in Older Adults With Cognitive Concerns: Pilot Cross-Sectional Study.

JMIR aging·2026
Same author

Stage-dependent dynamics of neuroinflammation across the Alzheimer's continuum.

Brain, behavior, and immunity·2026
Same author

Self-Reported Driving Behaviours and the Relationship With Sleepiness in Subjective Cognitive Decline and Mild Cognitive Impairment.

Journal of geriatric psychiatry and neurology·2026
Same journal

Breaking barriers: Enhancing access to dementia clinical trials in the United Kingdom-Insights from the Scientific Advisory Board of the Dame Barbara Windsor Dementia Goals Programme.

Alzheimer's & dementia : the journal of the Alzheimer's Association·2026
Same journal

Unveiling the procoagulant state in Alzheimer's disease: A novel PET imaging strategy.

Alzheimer's & dementia : the journal of the Alzheimer's Association·2026
Same journal

Estimated labor market outcomes of people progressing from preclinical to early-stage Alzheimer's disease in the United States.

Alzheimer's & dementia : the journal of the Alzheimer's Association·2026
Same journal

Amyloid exacerbates tau and alpha-synuclein pathologies, behavioral impairments, and neuroinflammation in a mixed dementia model.

Alzheimer's & dementia : the journal of the Alzheimer's Association·2026
Same journal

Multimorbidity burden and patterns associated with DeepBrainNet-derived brain-age gap in dementia-free older adults: A community-based study.

Alzheimer's & dementia : the journal of the Alzheimer's Association·2026
Same journal

Reply to "Shifting the emphasis of brain health literacy from individuals to systems to reduce inequalities".

Alzheimer's & dementia : the journal of the Alzheimer's Association·2026
查看所有相关文章

相关实验视频

Updated: Jan 7, 2026

Dried Blood Spot Collection of Health Biomarkers to Maximize Participation in Population Studies
07:20

Dried Blood Spot Collection of Health Biomarkers to Maximize Participation in Population Studies

Published on: January 28, 2014

37.1K

生物标志物 生物标志物

Aaron Kin Fu Lam1,2,3, Johannes C Michaelian2,4,5, Rachael Yu2,3

  • 1Woolcock Institute of Medical Research, Macquarie Park, NSW, Australia.

Alzheimer's & dementia : the journal of the Alzheimer's Association
|December 25, 2025
PubMed
概括
此摘要是机器生成的。

改变REM睡眠减速与老年人低粉样β 42/40水平有关. 这表明REM缓慢可能成为阿尔茨海默病神经病理学的早期生物标志物.

更多相关视频

Selecting Multiple Biomarker Subsets with Similarly Effective Binary Classification Performances
07:35

Selecting Multiple Biomarker Subsets with Similarly Effective Binary Classification Performances

Published on: October 11, 2018

7.9K
Ecotoxicological Methodologies to Evaluate Biomarkers at Different Scales in Neotropical Anurans
08:14

Ecotoxicological Methodologies to Evaluate Biomarkers at Different Scales in Neotropical Anurans

Published on: April 28, 2023

702

相关实验视频

Last Updated: Jan 7, 2026

Dried Blood Spot Collection of Health Biomarkers to Maximize Participation in Population Studies
07:20

Dried Blood Spot Collection of Health Biomarkers to Maximize Participation in Population Studies

Published on: January 28, 2014

37.1K
Selecting Multiple Biomarker Subsets with Similarly Effective Binary Classification Performances
07:35

Selecting Multiple Biomarker Subsets with Similarly Effective Binary Classification Performances

Published on: October 11, 2018

7.9K
Ecotoxicological Methodologies to Evaluate Biomarkers at Different Scales in Neotropical Anurans
08:14

Ecotoxicological Methodologies to Evaluate Biomarkers at Different Scales in Neotropical Anurans

Published on: April 28, 2023

702

科学领域:

  • 神经科学是一个神经科学.
  • 老年学是指老年学的学科.
  • 生物标志物发现发现

背景情况:

  • 改变睡眠神经生理学可能表明早期的大脑病理.
  • 之前没有研究将睡眠神经生理学与基于血液的神经退行生物标志物联系起来.
  • 研究了认知障碍老年人血液生物标志物和睡眠之间的关联.

研究的目的:

  • 为了确定基于血液的阿尔茨海默氏症和神经退行生物标志物是否与睡眠神经生理学相关.
  • 为了探索潜在的早期标志物大脑病理在老年人的认知问题.

主要方法:

  • 招募了50岁以上的成年人,有认知/情绪问题.
  • 参与者被分类为轻度认知障碍 (MCI) 或主观认知障碍 (SCI).
  • 测量了血生物标志物 (Aβ42/40,GFAP,NFL,pTau181) 和睡眠指标 (螺旋密度,泰达/三角形功率,REM减速) 通过多睡眠和血液测试.

主要成果:

  • 与SCI组相比,MCI组的Aβ42/40较低,GFAP,NFL,pTau181较高.
  • 甲基42/40与子密度和REM减缓有关.
  • GFAP与轴心密度相关. 轴心密度与轴心密度相关. GFAP与轴心密度相关.
  • 在对临床状态和年龄进行调整后,REM缓慢仍然是Aβ42/40的重要预测因素.

结论:

  • 快速睡眠缓慢是与血Aβ42/40.0相关的强有力的标志物.
  • 雷姆睡眠缓慢可能表明早期的神经病理变化,可能反映了粉样蛋白积累.
  • 需要进一步的纵向研究来确认睡眠神经生理学和AD生物标志物之间的因果关系.