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神经元经历IFNγ驱动的持续表观遗传变化和脑炎中的突触

Ghazal Shammas1, Margot Piccinno1, Kristof Egervari2

  • 1Department of Pathology and Immunology, University of Geneva, 1211 Geneva, Switzerland.

Neuron
|December 25, 2025
PubMed
概括

细胞毒性T细胞向中枢神经系统疾病中的神经元,导致持续的突触损伤. 神经元中一种新的干扰素- (IFNγ) 驱动的表观遗传程序导致持续的突触,影响神经炎症.

关键词:
染色体重塑 染色体重塑 的方法细胞毒性T细胞细胞毒性脑炎是一种脑炎.多种主题的多种主题.神经元神经元的神经元非细胞分解性病毒清除率发生突触损失.转录适应的转录适应

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In the CNS, neurogenesis, the birth of new neurons from stem cells, is limited to the hippocampus in adults. In other regions of the brain and spinal cord, neurogenesis is almost non-existent due to inhibitory influences from neuroglia, especially oligodendrocytes, and the absence of growth-stimulating cues. The myelin produced by oligodendrocytes in the CNS inhibits neuronal regeneration. Furthermore, astrocytes proliferate rapidly after neuronal damage, forming scar tissue that physically...
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