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Updated: Jan 7, 2026

Optimized Analysis of In Vivo and In Vitro Hepatic Steatosis
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一个ATF3诱导器通过AMPK和PKA途径改善了与代谢功能障碍相关的脂肪性肝病

Ching-Feng Cheng1,2,3, Ruey-Bing Yang2, Wen-Ting Chen1

  • 1Department of Pediatrics, Taipei Tzu Chi Hospital, Buddhist Tzu Chi Medical Foundation, New Taipei City 23142, Taiwan.

International journal of molecular sciences
|December 30, 2025
PubMed
概括

在与代谢功能障碍相关的脂肪性肝病 (MASLD) 中,ST32db可降低脂质积累. 这种化合物激活肝脏的AMPK和PKA通路,为MASLD提供潜在的治疗益处.

关键词:
通过AMP激活的蛋白激酶激酶被AMP激活.马斯尔德 马斯尔德是什么意思激活转录因子3诱导剂蛋白酶A是一种蛋白酶.

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