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Updated: Jan 7, 2026

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通过促进线粒体功能障碍,Ncor1的Nsun2介导的m5C甲基化加剧了败血症引起的心肌病

Chan Chen1, Qing Liu1, Junmei Xu1

  • 1Department of Anesthesiology, The Second Xiangya Hospital, Central South University, Changsha 410011, Hunan, China.

Free radical biology & medicine
|January 1, 2026
PubMed
概括

No abstract available in PubMed .

关键词:
心肌细胞亡线粒体裂变没有.没有.败血症引起的心肌病 (SIC)m5CRNA甲基化

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The mitochondrial electron transport chain (ETC) is the main energy generation system in the eukaryotic cells. However, mitochondria also produce cytotoxic reactive oxygen species (ROS) due to the large electron flow during oxidative phosphorylation. While Complex I is one of the primary sources of superoxide radicals, ROS production by Complex II is uncommon and may only be observed in cancer cells with mutated complexes.
ROS generation is regulated and maintained at moderate levels necessary...
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