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Updated: May 2, 2026

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微细胞HVCN1缺陷通过增强微细胞迁移和神经保护来改善SOD1G93AALS小鼠的运动和生存

Fan Wang1, Ke-Yu Zhang1, Lang-Jian Zhu2,3

  • 1Department of Pathology of Sir Run Run Shaw Hospital and Department of Human Anatomy, Histology and Embryology, System Medicine Research Center, NHC and CAMS Key Laboratory of Medical Neurobiology, Zhejiang University School of Medicine, Hangzhou, Zhejiang, China.

Advanced science (Weinheim, Baden-Wurttemberg, Germany)
|January 4, 2026
PubMed
概括

No abstract available in PubMed .

关键词:
在HVCN1/HV1肌缩侧面硬化症微质细胞迁移情况神经保护细胞发生

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Huntington Disease l: Introduction01:21

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Huntington disease or HD is a progressive, fatal neurodegenerative disorder inherited in an autosomal dominant pattern.PathophysiologyIt is caused by expansion of the CAG trinucleotide repeat in the HTT gene on chromosome 4 (4p16.3), producing an abnormal huntingtin protein with an expanded polyglutamine tract. This misfolded protein disrupts cellular function, leading to neuronal death. Normal alleles have ≤26 repeats, 27–35 are intermediate (risk of expansion), 36–39 show...
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